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Symposium

日本-韓国神経科学学会合同シンポジウム:Molecular Mechanisms underlying Parkinson Disease
Joint Symposium of the Japan Neuroscience Society and the Korean Society of Brain and Neuroscience:Molecular Mechanisms underlying Parkinson Disease

開催日 2014/9/12
時間 9:00 - 11:00
会場 Room C(502)
Chairperson(s) 望月 秀樹 / Hideki Mochizuki (大阪大学医学系研究科神経内科学 / Department of Neurology, Osaka University Graduate School of Medicine, Japan)
Young Jun Oh (Department of Systems Biology, Yonsei University College of Life Science and Biotechnology, Korea)


Opening Remarks for the First Korea-Japan Neuroscience Symposium

  • S2-C-1-1
  • 髙橋 良輔 / Ryosuke Takahashi:1 
  • 1:京都大学大学院医学研究科・臨床神経学 / Department of Neurology, Kyoto University Graduate School of Medicine, Japan 


Lysosomal dysfunction is the key factor for propagation of synucleinopathy

  • S2-C-1-2
  • Seung-Jae Lee:1 Eun-Jin Bae:1 Na-Young Yang:1 Cheol-Soon Lee:1 He-Jin Lee:1 Eliezer Masliah:2 Pablo Sardi:3 
  • 1:Konkuk University, Seoul, Korea 2:University of California, San Diego, La Jolla, CA, USA 3:Genzyme, a Sanofi Company, Framingham, MA, USA 


Function of transient receptor potential vanilloid 1 in Parkinson’s disease

  • S2-C-1-3
  • Byung Kwan Jin:1 
  • 1:Kyung Hee University ,Korea 



Structure and function of α-synuclein

  • S2-C-1-5
  • 望月 秀樹 / Hideki Mochizuki: 
  • 1:大阪大学医学系研究科神経内科学 / Department of Neurology, Osaka University Graduate School of Medicine, Japan 

異常αシヌクレインの広がりが病気の進行を説明する
Spreading of pathological alpha-synuclein explain the disease progression

  • S2-C-1-6
  • 長谷川 成人 / Masato Hasegawa:1 
  • 1:東京都医学総合研究所 / Tokyo Metropolitan Institute of Medical Science, Japan 

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