日本－韓国神経科学学会合同シンポジウム：Molecular Mechanisms underlying Parkinson Disease
Joint Symposium of the Japan Neuroscience Society and the Korean Society of Brain and Neuroscience：Molecular Mechanisms underlying Parkinson Disease

Opening Remarks for the First Korea-Japan Neuroscience Symposium

• S2-C-1-1
• 髙橋 良輔 / Ryosuke Takahashi:1　
• 1:京都大学大学院医学研究科・臨床神経学 / Department of Neurology, Kyoto University Graduate School of Medicine, Japan　

Lysosomal dysfunction is the key factor for propagation of synucleinopathy

• S2-C-1-2
• Seung-Jae Lee:1　Eun-Jin Bae:1　Na-Young Yang:1　Cheol-Soon Lee:1　He-Jin Lee:1　Eliezer Masliah:2　Pablo Sardi:3　
• 1:Konkuk University, Seoul, Korea　2:University of California, San Diego, La Jolla, CA, USA　3:Genzyme, a Sanofi Company, Framingham, MA, USA　

Function of transient receptor potential vanilloid 1 in Parkinson’s disease

• S2-C-1-3
• Byung Kwan Jin:1　
• 1:Kyung Hee University ,Korea　

Impaired autophagic flux is linked to calcium-dependent dopaminergic neuronal death

• S2-C-1-4
• Young Jun Oh:1　
• 1:Yonsei University, Korea　

Structure and function of α-synuclein

• S2-C-1-5
• 望月 秀樹Hideki Mochizuki　
• 1:大阪大学医学系研究科神経内科学 / Department of Neurology, Osaka University Graduate School of Medicine, Japan　

異常αシヌクレインの広がりが病気の進行を説明する
Spreading of pathological alpha-synuclein explain the disease progression

• S2-C-1-6
• 長谷川 成人 / Masato Hasegawa:1　
• 1:東京都医学総合研究所 / Tokyo Metropolitan Institute of Medical Science, Japan